Abstract
Impairment in working memory (WM) and emotional processing are features of psychiatric disorders. Although cognitive deficits may be driven by synaptic/neuritic alterations, in vivo evidence is lacking. Recent studies confirmed neuritic architecture (NAR) alterations in vivo in schizophrenia using Neuritic-Orientation-Dispersion-and-Density Imaging (NODDI). To identify biological mechanisms underlying brain activity, we characterized relationships between NAR, BOLD (Blood-Oxygen-Level-Dependent) activation and cognition using human in vivo imaging.
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