Abstract
Neonatal neuronal exuberance and its retention following neonatal brain damage have been demonstrated in a number of species but not in humans. The purpose of the present ongoing study is to determine if there is any evidence of neonatal neuronal exuberance and its retention following damage to the CNS in the human. Of equal concern is the determination of the neurological mechanisms underlying abnormal movement and reflex development in children with cerebral palsy.
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