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https://doi.org/10.1093/cvr/7.3.370
Copy DOIJournal: Cardiovascular research | Publication Date: May 1, 1973 |
Citations: 2 |
Myofibrils were prepared from the left ventricles of normal dogs, of dogs after cardiac denervation, and of dogs in low output cardiac failure after cardiac homotransplantation. The ATPase activity of the myofibrils in the presence and absence of calcium was measured. It was found that cardiac denervation and cardiac homotransplantation leading to cardiac failure did not alter the intrinsic ATPase activity of myofibrils in either the presence or absence of calcium. Although this study does not eliminate a defect in excitation-contraction coupling in cardiac failure secondary to transplantation, it indicates that there is no primary defect in the intrinsic ATPase activity of cardiac myofibrils from transplanted hearts in failure.
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