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Open Accesshttps://doi.org/10.1158/2767-9764.crc-24-0069
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Cite Journal: Cancer research communications |  Publication Date: Sep 1, 2024 |
 License type: cc-by |
Prostate cancers that escape therapy targeting the androgen receptor signaling pathways via phenotypic plasticity are currently untreatable. Our study identifies MYBL2 as a MR-TF in phenotypic plastic prostate cancer and implicates CDK2 inhibition as a novel therapeutic target for this most lethal subtype of prostate cancer.
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