Abstract
While many advances have been made in the area of kidney stone treatment and disease, relatively very little is known about the process within the kidney that initiates stone formation. Despite recent advances in medical treatment, the recurrence can be reduced only 50%. Therefore identifying cellular processes that can initiate stone formation is important to prevent stone recurrence. We have previously demonstrated that oxalate-induced reactive oxygen species (ROS) mediated cell injury appears to play a significant role in kidney stone formation.
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