Abstract
Transcranial alternating current stimulation (tACS) modulates neural oscillatiory activity and excitability in the primary motor cortex (M1). Previous studies have found that these effects can last for more than an hour and an involvement of N-methyl-D-aspartate receptor (NMDAR) mediated synaptic plasticity has been suggested. However, the cortical mechanisms underlying tACS after-effects are not fully understood. In the present study 20 Hz beta tACS was applied to M1 after participants received either an NMDAR antagonist (dextromethorphan) or a placebo and the effects on cortical beta oscillations and excitability were explored.
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