Abstract
Depression is characterized by both reduced positive affect (PA) and enhanced negative affect (NA). Teasdale’s Differential Activation Hypothesis suggests that depressed individuals have increased cognitive reactivity during episodes of depressed mood. Understanding which neural networks are activated during depressed mood in major depressive disorder (MDD) could elucidate the underlying pathophysiology for treatment-resistant depression. One method for characterizing these neural networks is through using magnetoencephalography (MEG) during a mood induction paradigm.
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