Abstract
Diabetes and its complications including renal impairment have been suggested to be consequences of increased oxidative stress. Glucose-6-phosphate dehydrogenase (G6PD) is an enzyme of pentose monophosphate shunt which is responsible for protecting from dangerous oxidative metabolites. At the same time, G6PD deficiency is the most common enzyme deficiency in humans which leaves these individuals at high risk of oxidative stress and at high risk of diabetes and later complications of diabetes including nephropathy.
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