Abstract
234.0±46.9‰). The serum MDA level in the STZ group was significantly elevated, which was not reversed by addition of rebamipide. The expression of gastric HO-1 in the STZ group was significantly increased, and this increase was supressed by rebamipide supplementation. The expression of gastric c-kit was not affected by the administration of rebamipide. Conclusion. Administration of rebamipide improved diabetic gastric motility disorders through protection against HO-1 reactive gastric oxidative stress.
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