Abstract
Quantitative behavioral tests showed that unilateral bulbar lesions made in anesthetized cats, damaging nucleus gigantocellularis (NGc), nucleus paragigantocellularis dorsalis (Pgd), and/or the magnocellular tegmental field (FTM), increased latency and threshold voltage for escape from electric shock to the feet. Motor function, visual orientation, and orientation to sound were normal. It is hypothesized that NGc, Pgd, and FTM may constitute a functional complex, operating such that adequate unilateral damage to this field significantly disrupts input into the midline thalamic area.
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