Abstract
Recent experiments in epithelial wound healing have demonstrated the necessity of MAPK activation for coordinated cell movement after damage. This MAPK activity is characterized by two wave-like phenomena. One MAPK “rebounding wave” that originates immediately after injury, propagates deep into the cell layer, and then regresses back to the wound interface. The second MAPK wave is a slow developing, sustained wave that propagates from the wound interface. Experimentalists have suggested that the first wave is originated by reactive oxygen species (ROS) generated at the time of injury.
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