Abstract
G A A b st ra ct s pre-central, post-central and anterior cingulate gyrus, insula and thalamus in all groups. However in patient group we observed activation in right lingual gyrus, left fusiform gyrus, and bilateral in medial frontal gyrus, thalamus, cingulate gyrus, pons, cerebellum, SMA and fronto-parietal opercula but there was lower activation of right inferior parietal, bilateral middle frontal gyrus, right insula, right primary motor area, in comparison to control. Conclusion: Patients with achalasia had increased activation in motor cortex suggesting involvement of primary motor cortex that may contribute to occurrence of dysphagia. Increased activation in inferior temporal gyrus and lingual gyrus in patients with achalasia suggest more urge, intent and planning for initiation of swallow. The results suggest neurodegenerative changes in the myenteric plexus in the esophagus.
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