Abstract
Background and Aims : Reactive oxygen species overproduction driven by up-regulated NADPH oxidase (Nox) plays a major role in atherosclerosis. Hypoxia-induced miR-210-3p has been largely implicated in human malignancies as key regulator of oncogenic mechanisms. The role of miR-210-3p in atherogenesis is yet to be discovered. This study aimed at investigating the potential role of miR-210-3p in mediating Nox up-regulation in atherosclerosis.
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