Abstract
Background and Aims: Abdominal aortic aneurysms (AAAs) are highly lethal diseases without effective clinical predictors and therapeutic targets. Vascular microcalcification as detected by fluorine-18-sodium fluoride (18F-NaF) has recently been recognized as a valuable indicator in predicting of atherosclerotic plaque rupture and AAA expansion. However, whether vascular microcalcification is involved in the pathogenesis of AAA remains elusive.
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