Abstract
Classically, septic cardiomyopathy has been defined as a decrease in the left ventricular ejection fraction in the presence of sepsis.1,2 However, assessment of ejection fraction has shown to have low sensitivity in the presence of shock because of the reduced afterload associated with it, giving increase to a false “normal” left ventricular ejection fraction. Consequently, efforts to find alternative surrogates to assess left ventricular systolic and diastolic function in sepsis are underway. In the context of imperfect surrogates, the association between ventricular dysfunction and mortality is highly variable, reinforcing the notion that a clear definition of septic cardiomyopathy is lacking.
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