Abstract
PROSTAGLANDINS have been suggested as mediators of inflammatory reactions because they increase vascular permeability1,2, are found in inflammatory exudates3, are released during antigen-antibody reactions4 and have leucotactic properties5. For this reason and because lysosomes have been assigned an important role in the pathogenesis of diverse tissue injury reactions6, we have studied the effect of prostaglandins on lysosomal fragility.
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