Abstract
Dr. John H. Menkes's review of the factors responsible for the lowering of the spinal fluid sugar in bacterial and tuberculous meningitis1 presents evidence that the primary factors for the low spinal fluid sugar in bacterial meningitis may be a combination of increased utilization of glucose by the brain, combined with a defective glucose transport. We2 presented evidence over 30 years ago that the lowering of the spinal fluid sugar in tuberculous meningitis was not due predominantly to utilization of the glucose by either bacteria or leukocytes:
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