Abstract
Hydrocephalus that develops after subarachnoid hemorrhage is of particular interest in relation to some forms of dementia. Various studies, including examination of cerebrospinal fluid (CSF) circulation, X-ray CT, and monitoring of cerebral blood flow and intracranial pressure, have been performed. However, little is known about the changes of oxygen metabolism, which is closely related to brain function. Cerebral oxygen metabolism and blood flow were studied by means of positron emission CT (PET) to elucidate the involvement of cerebral oxygen metabolism in the pathophysiology of normal- and high-pressure hydrocephalus after subarachnoid hemorrhage.
Published Version
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