Abstract
Background and Aims: Infectious diseases affect lipid homeostasis, reflecting the role of lipids in host pathogen interactions. In sepsis patients, HDL and LDL-cholesterol levels decrease, while triglycerides and VLDL increase. High cholesterol could suggest an evolutionary advantage of this condition. We hypothesize that alterations of cellular and systemic lipid metabolism alter the course of bacterial infections and the associated host immune responses.
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