Abstract
The acute effect of airway stimulation by capsaicin is a cough reflex. The asthma defined by a chronic bronchial inflammatory state can include neurogenic inflammation. Asthma-like disorders are possible following recurrent exposure to capsaicin. The capsaicin acts by stimulating the intrabronchial vanilloid receptors and causes a nerve impulse which can induce neurogenic inflammation mediated by the peptide neuromediators. Hyperresponsiveness to capsaicin defines sensitive hyperreactivity which is indissociable from the bronchial hyperreactivity in several forms of asthma. The impact of prolonged exposure was not studied, the asthma worsened and would even be caused by the neurosensory chemical stimulants. Transverse studies, in particular at the workplace, are necessary to screen this original entity.
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