Abstract
The limited capacity for division exhibited by normal animal cells in culture is well documented and a striking contrast to the unlimited division potential of abnormal tumor-derived or virus- or carcinogen-transformed cells. In 1965 Hayflick proposed that the limited division of normal cells was a manifestation of aging at the cellular level, and we have studied normal human cells, particularly fibroblasts, as a model for in vitro aging, attempting to understand the basic mechanisms involved in normal growth control.
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