Abstract
Background and Aims: Type 2 diabetes mellitus (T2DM) is one of the major risk factors for cardiovascular complications. Reduced relaxation of blood vessels from diabetic patients could be result of different expression and/or function of smooth muscle potassium (K) channels. Thus, the objective of our study was to investigate differences in the involvement of voltage-gated K (Kv) channels in the effect of pinacidil on human saphenous veins (HSV) obtained from patients with/without T2DM.
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