Abstract
In this study the authors [1] have looked in great detail into the potential pathophysiology of abnormal enteric protein loss (AEPL), also known as protein-losing enteropathy after Fontan-type operations. Designed as a cross-sectional study during outpatient visits, mesenteric and celiac artery blood flows were measured by ultrasound and markers for inflammation, liver function, coagulopathy, and glycosylation defects, as well as neurohormonal levels were determined, thereby covering the current pathophysiologic theories.
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