Abstract
Chorioamnionitis is associated with just over 1/3 of PTBs. Historically, it is thought to be a result of ascending infection with relatively low-virulence organisms. However, pathophysiology studies in humans suggest that it results from focal membrane colonization. Here, we aimed to determine if sterile intraamniotic injection (IAI) of IL1β, LPS, or Ureaplasma parvum (UP) induced-chorioamnionitis in primates is associated with alterations in the resident metagenomic composition and its function.
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