Abstract
Analysis of intervertebral disc regeneration after disc height restoration by posterolateral spinal fusion with lumbar distraction of 68 patients diagnosed with degenerative lumbar instability.
Highlights
Degenerative disc disease (DDD) is the main cause of low back pain [1,2]
Lumbar spine instability formed on the basis by disc degeneration is considered one of the most important factors of spinal pain and one of the most common indications for fusion surgery following ineffective conservative treatment [8]
It is clear that posterior distraction of the lumbar spine on a straight Harrington rod will diminish lumbar lordosis and result in flat back syndrome [26]
Summary
Degenerative disc disease (DDD) is the main cause of low back pain [1,2]. The most important factor involved in the development of DDD is the vertical load of spine, which can activate enzymatic processes facilitating discs degenerations, in addition to the purely direct (increased fluid stress and hydrostatic pressure) and indirect (decreased permeability and nutrition) mechanical effects. [3] The repetitive flexion movements and certain genetic characteristics may play an important role in DDD. [4].One of the most common conditions accompanying DDD is segmental instability of the spine. Degenerative disc disease (DDD) is the main cause of low back pain [1,2]. The most important factor involved in the development of DDD is the vertical load of spine, which can activate enzymatic processes facilitating discs degenerations, in addition to the purely direct (increased fluid stress and hydrostatic pressure) and indirect (decreased permeability and nutrition) mechanical effects. One of the most common conditions accompanying DDD is segmental instability of the spine. The pathogenesis of segmental instability was first described by Kirkaldy-Willis [5]. He divided the evolution of the disease into three stages called temporary dysfunction, unstable phase and secondary stabilization. Early signs of disc degeneration can only be discovered by MRI. The abnormality compared to normal discs is a low signal in the nucleus pulpous without decreased disk height or changes in the contour of the annulus
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