Abstract
AbstractApart from acute coronary syndrome, increased troponin represents 40 % of requests. In acute myocardial infarction (AMI), the increase in cardiac troponin is linked to cardiomyocyte necrosis. But any reversible or irreversible cardiomyocyte injury can be accompanied with an increase in cardiac troponin. It is important to know how to distinguish causes without any real deleterious impact, such as intense physical exercise, from the numerous pathological causes where the high value of cardiac troponin constitutes a prognostic factor : renal failure, pulmonary embolism, sepsis, non-cardiac surgery. The increase in cardiac troponin T alone in various muscular disorders seems more and more frequent and leads us to question the necessity of having the dosage of both cardiac troponin T and cardiac troponin l.
Published Version
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