Abstract
Background and Aims : A central role of smooth muscle cells (SMCs) in atherosclerosis has recently evolved implying genetic and causal links in the disease. Here, we hypothesized that coronary artery disease (CAD)-related gene polymorphisms influence SMC function in atherosclerosis and investigated the role of genes harboring such variants in SMCs in vitro.
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have