Abstract
It has been shown that under the action VV-GMCSF-Lact, the death of glioblastoma cells occurs mainly through the apoptotic pathway. When analyzing the complexes of Smac protein with the apoptosis inhibitor XIAP in human glioblastoma cells, it was found that in the cells most sensitive to VV-GMCSF-Lact, Smac level was increased after incubation with the virus. At the same time, overall level of XIAP was higher in more resistant cells.
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