Abstract
Administration of 2,4-dimethoxyphenylbiguanide to rats with experimental myocardial infarction decreased the activity of cardiomyocyte-cytolysis marker enzymes in rat blood serum and the extent of free-radical oxidation as estimated from biochemiluminescence parameters and activities of aconitase, superoxide dismutase, and catalase in the heart and blood serum as compared with pathology that was evidently related to a decreased degree of reciprocal antioxidant system mobilization. The results could provide a basis for further studies of 2,4-dimethoxyphenylbiguanide as a promising antioxidant and cardioprotector.
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