Abstract
Inflammatory reaction in neurological diseases.
Highlights
In acute critical central nervous system (CNS) diseases, the study by K.-W
In acute critical CNS diseases, such as delayed deterioration associated with vasospasm after subarachnoid hemorrhage (SAH), ischemic stroke, spontaneous intracerebral hemorrhage (ICH), and traumatic brain injury (TBI), recent evidences show that inflammation may be a potential target for therapy
To accelerate the process of translating this information to clinical applications, a number of important issues must be addressed such as their ability to consistently detect characteristic cerebral deficits in individuals with neurological degenerative diseases, the relationships of cerebral injuries to clinical symptoms and genetic characteristics, and the degree to which these injuries respond to different therapies
Summary
In acute critical CNS diseases, the study by K.-W. Wang et al used traumatic brain injury (TBI) model to determine whether simvastatin combined with an antioxidant could attenuate cerebral vascular endothelial inflammatory response after traumatic brain injury in rat. Hung-Chen Wang,1 Cheng-Hsien Lu,2,3 Kuang-I Cheng,4 and Jason Cheng-Hsuan Chiang5 Inflammatory reaction in the central nervous system (CNS) is recognized to be a feature of all neurological disorders.
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