Abstract
There are many biochemical elements implicated with the inflammation mediators and markers and this review show their identities and their atherosclerotic effects. Cytokines identified as participants in the mechanisms of atherogenesis were first discovered as involved in the process of natural (or innate) and specific (or acquired) immunity, responsible for defending against foreign organisms such as viral or bacterial agents. They are protein hormones produced mainly by mononuclear phagocytes (monocins) in direct response to microbes, or originated from T lymphocytes (lymphocins) when stimulated by antigen, as part of specific immunity. T lymphocytes produce several cytokines that primarily serve to regulate the activation, growth and differentiation of the various lymphocyte populations. Other cytokines derived from the T lymphocyte work primarily in the activation and regulation of inflammatory cells, such as mononuclear phagocytes, neutrophils and eosinophils. Thus, cytokines derived from T lymphocyte are effector molecules of cell-mediated immunity, and are also responsible for communications between the cells of the immune and inflammatory systems.
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