Abstract
In field plot experiments, Mycocentrospora acerina delayed emergence and caused ‘damping-off’ and death of emerged carrot seedlings, substantially reducing the population density. Surviving plants were often stunted and usually senesced earlier than healthy ones. The fungus was one of the main primary colonizers of the root surfaces where it developed extensively as hyaline mycelium and then formed pigmented mycelium and numerous chlamydospores as the tap root developed. These remained attached to the roots during harvest and were the main source of inoculum for infection during storage. Leaf lesions, bearing conidia of M. acerina , were also observed towards the end of the growing season but appeared to be relatively unimportant in the build up of the fungus in the field or in storage.
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