Abstract
Systemic amyloidosis is marked by accumulation of amyloid deposits in various tissues throughout the body. In most cases, impairment of organ function by these deposits leads to morbid and complex pathological conditions. Primary systemic amyloidosis (AL) is caused by accumulation of monoclonal immunoglobulin light‐chains (LC), which are overproduced by plasma cells [1]. AL is the most prevalent form of systemic amyloidosis. Secondary systemic amyloidosis (AA) can be recognized by the accumulation of serum amyloid A protein, a process that is thought to be a hazardous side‐effect of chronic inflammation [2].
Full Text
Published Version (
Free)
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call