Abstract
Chronic granulomatous disease (CGD) is a rare inherited immunodeficiency characterized by microbicidal dysfunction due to a defect in nicotinamide adenine dinucleotide phosphate oxidase in neutrophils. Patients with CGD have abnormalities in cytokine production and apoptosis leading to prolonged inflammation, which is related to the susceptibility to autoimmune or inflammatory diseases such as sarcoidosis, Crohn’s disease, systemic lupus erythematosus, Behcet’s disease, and Henoch‐Schonlein purpura. 1
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