Abstract
Purpose: Osteoarthritis (OA) is the most common age-related musculoskeletal disease with no curative treatment so far. Recently, it has been observed that bone remodeling is accompanied by an increase in the number of medullary adipocytes at the expense of osteoblasts in age-associated diseases. Some studies suggest that adipocytes from the bone marrow could play a role in bone homeostasis, yet their role in OA is to be discovered. In the present study, we hypothesize that bone marrow adipocytes (BMAds) are crucial cell partners in the control of bone homeostasis and that their physiological functions are deregulated in OA.
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