Abstract
Introduction. Left ventricular outflow obstruction might be part of the pathophysiological mechanism of Tako-tsubo cardiomyopathy. This obstruction can be masked by Tako-tsubo cardiomyopathy and diagnosed only by followup. Case Presentation. A 70-year-old female presented with Tako-tsubo cardiomyopathy and masked obstructive hypertrophic cardiomyopathy at presentation. Conclusion. Tako-tsubo cardiomyopathy typically presents like an acute MI and is characterized by severe, but transient, regional left ventricular systolic dysfunction. Prompt evaluation of the coronary status is, therefore, mandatory. The prognosis under medical treatment of heart failure symptoms and watchful waiting is favourable. Previous studies showed that LVOT obstruction might be part of the pathophysiological mechanism of TCM. This paper supports this theory. However, TCM may also mask any preexisting LVOT obstruction.
Highlights
Left ventricular outflow obstruction might be part of the pathophysiological mechanism of Tako-tsubo cardiomyopathy
Tako-tsubo cardiomyopathy typically presents like an acute MI and is characterized by severe, but transient, regional left ventricular systolic dysfunction
Previous studies showed that LVOT obstruction might be part of the pathophysiological mechanism of tsubo cardiomyopathy (TCM)
Summary
Tako-tsubo cardiomyopathy (TCM) is an acute cardiac syndrome of unknown etiology characterized by severe but transient systolic dysfunction of the apical and/or mid segments of the LV mimicking myocardial infarction in the absence of obstructive coronary artery disease [1, 2]. This form of contractile dysfunction is typically transient and reversible within days or weeks [3, 4]. There was a pressure gradient below the level of the aortic valve between the aorta and the left ventricle
Sign-in/Register to view highlights & summary Sign-in/Register to access full text options 
Free) 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call
