Abstract
How can we improve outcomes of chlamydia control programmes?
Highlights
Chlamydia can cause salpingitis, which can be either symptomatic (pelvic inflammatory disease) or asymptomatic, and its sequelae, ectopic pregnancy and tubal factor infertility.[1] Given highly effective therapy and excellent diagnostic tests, chlamydia control programmes to reduce and prevent adverse reproductive consequences have been initiated widely
The obvious question is why is there excess risk for disease associated with chlamydia infection after diagnosis and treatment? With ectopic pregnancy and tubal factor infertility, the obvious answer is that these reflect previous Fallopian tube damage, which can occur with or without a previous symptomatic pelvic inflammatory disease episode
Why the excess risk of pelvic inflammatory disease? The intuitive answer is that chlamydia infection is a surrogate for other sexually transmitted infections that cause pelvic inflammatory disease
Summary
Chlamydia can cause salpingitis, which can be either symptomatic (pelvic inflammatory disease) or asymptomatic, and its sequelae, ectopic pregnancy and tubal factor infertility.[1] Given highly effective therapy and excellent diagnostic tests, chlamydia control programmes to reduce and prevent adverse reproductive consequences have been initiated widely. In The Lancet Infectious Diseases, Bethan Davies and colleagues[2] present a careful evaluation and thoughtful discussion of the risks of reproductive complications in a nationally representative sample of Danish women after being tested (and presumably treated) for chlamydia infection.
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