Abstract
A track structure model has been applied to the assessment of the carcinogenic risk of inhaled alpha-emitting radon decay products in bronchial tissue. Dose, fluence, LET-spectrum, dose-average LET, and track length distribution were calculated for different bronchial airway generations and sensitive cells located at varying depth in bronchial epithelium. Utilizing experimental radiobiological information, the radiation risk at a given depth in bronchial tissue is expressed by the probabilities for cellular survival, cell killing, mutation and transformation, and the joint probability for cancer induction. These calculations have shown that the lung cancer incidence per unit dose is a function of dose, questioning the linear extrapolation to low doses for high LET radiations.
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