Abstract
Bidirectional relationships between inflammation and metabolism may contribute to the pathophysiology of schizophrenia. Acoustic startle latency, the time between a startling stimulus and the reflexive response, is a putative index of processing speed and is slower in patients with schizophrenia than controls. Increased inflammation is associated with slowing of startle latency in patients. No one has investigated whether metabolic dysfunction may also contribute to slowing of latency in schizophrenia.
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