Abstract
SummarySevere hemorrhagic diatheses resulting from insufficient or delayed thromboplastin formation may have varied causes : (1) Antihemophilic globulin (A.H.G.) deficiency in classic hemophilia (hemophilia type A) in men or homozygote women (daughters of a hemophilic type A and carrier of the antihemophilic gene type A). (2) Plasma Tromboplastin Component (P.T.C.) deficiency (Christmas disease or hemophilia type 15). (3) Plasma Thromboplastin Antecedent (P.T.A.) deficiency (hemophilia type C). (4) Combined antihemophilic globulin and Plasma Thromboplastin Component deficiency. (5) A circulating anticoagulant may complicate the A.H.G. or P.T.C, deficiency and aggravate these conditions. (6) Presence of a circulating anticoagulant in non-hemophilic men. (7) In women, the presence of a circulating anticoagulant which may or may not be related to a recent pregnancy. (8) Antihemophilic globulin deficiency in women not belonging to a hemophiliac family. This condition is usually associated with a vascular defe...
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