Abstract
s1 May 1968Goiter and Hypothyroidism.Carl E. Cassidy, M.D., Richard L. Eddy, M.D., Edwin B. Astwood, M.D., F.A.C.P.Carl E. Cassidy, M.D.Search for more papers by this author, Richard L. Eddy, M.D.Search for more papers by this author, Edwin B. Astwood, M.D., F.A.C.P.Search for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-68-5-1143_3 SectionsAboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail ExcerptNontoxic goiter, a compensatory phenomenon, reflects a defect in the synthesis of thyroxine. The deficiency stimulates secretion of thyroid-stimulating hormone, enlargement of the thyroid ensues, the daily requirement of thyroxine is met, and the only abnormality that results is the "simple" goiter. We suspected that the compensation should not always be so complete and that goiter and hypothyroidism should sometimes coexist. The suspicion was confirmed by finding 234 patients during a 13-year period.The frequency of the signs and symptoms was tabulated. Both the protein-bound iodine (PBI) and the 24-hr131I uptake by the thyroid were carried out in 200... This content is PDF only. To continue reading please click on the PDF icon. Author, Article, and Disclosure InformationAffiliations: Boston, Massachusetts PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics Cited by2,2?-Diphenyl-?3,3?-bi-3H-indole-1,1?-dioxide: Competitive demethylation and redox reactions 1 May 1968Volume 68, Issue 5Page: 1143-1144KeywordsHypothyroidismProteinsSigns and symptomsThyroidThyroid-stimulating hormoneThyroxine ePublished: 1 December 2008 Issue Published: 1 May 1968 PDF downloadLoading ...
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