Abstract
Glutamine synthetase (GS) activity was studied in newborn mice homozygous for radiation-induced lethal albino alleles that cause multiple biochemical deficiencies. GS was found to be decreased in the liver of both homozygous mutants studied, whereas enzyme levels were normal in the eye and brain. The results support the interpretation that the neonatal lethal deletion alleles are mutations of a regulatory genome that normally controls the activities of multiple enzymes.
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