Abstract
Recent studies examining the role of dendritic cell (DC) subsets demonstrate that myeloid DCs (mDCs) promote allergen-induced AHR, while plasmacytoid DCs (pDCs) are required to induce regulatory T cell activity in models of tolerance. Herein, we explore the extent to which differential recruitment of pulmonary and lymph node (LN) DC populations influences airway responses using mice resistant (C3H/HeJ) or susceptible (A/J) to the development of allergen-induced asthma.
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