Abstract
We propose a new schema of genetic regulation in bacteria. This schema is based on the following two points: 1. a) All nonsense mutations would not have the same probability of preventing the translation. 2. b) There would be interactions of competitive type between the short m-RNA and the homologous long m-RNA. According to this hypothesis, the regulator and operator genes appear only as simple structural genes. Each structural gene may have a regulatory, coordinated function on all the not necessarily linked genes which control the synthesis of the enzymes of a metabolic pathway. The result is a new conception of the operon. Repression and induction are regulated by the same mechanism. This new hypothesis proves to be in agreement with the observations that support the model proposed by Jacob and Monod.
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