Abstract
Background and Aims: Aim: Pathological vascular remodeling is the underlying cause of main cardiovascular diseases (CVD) including atherosclerosis and abdominal aortic aneurysm (AAA). Diminished vascular smooth muscle cell (VSMC) content is a key mechanism of atherosclerotic plaques and AAA instability. We hypothesized that Galectin-1 (Gal-1) could exert protective effects in atherosclerosis and AAA as previously demonstrated in different chronic and acute inflammatory diseases.
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