Abstract
The rostral ACC (rACC) and medial thalamic parafascicular nucleus (Pf) are implicated in the processing and modulation of pain affect. Anatomical and physiological evidence suggests that Pf supplies glutamatergic afferents to rACC, and nociceptive-reactive neurons in Pf function as a critical relay of nociceptive input to rACC. In the present study, we evaluated the interaction between the rACC and Pf in suppressing the vocalizations of the rat that occur following application of noxious tailshock (vocalization afterdischarges, VADs).
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