Abstract
Perennial ryegrass is overseeded in bermudagrass and Kentucky bluegrass to improve turf quality, but selective control may be warranted for transition back to monostand turfgrass. Flucarbazone–sodium controls perennial ryegrass in bermudagrass and Kentucky bluegrass, but the physiological basis of selectivity has received limited investigation. Greenhouse and laboratory experiments were conducted to evaluate efficacy, absorption, translocation, and metabolism of flucarbazone–sodium in these grasses. Flucarbazone–sodium reduced perennial ryegrass shoot mass from the nontreated an average ≈ 22 times and 3 times more than bermudagrass and Kentucky bluegrass at 4 wk after treatment, respectively. In laboratory experiments, foliar and root absorption of14C–flucarbazone–sodium were similar among species. Bermudagrass distributed ≈ 25% more foliar-absorbed14C to nontreated shoots than Kentucky bluegrass and perennial ryegrass. From root applications, all grasses averaged 84% distribution of14C to shoots. Bermudagrass and Kentucky bluegrass metabolized 100% and 74% of14C–flucarbazone–sodium at 1 d after treatment (DAT), whereas perennial ryegrass metabolism measured 44, 58, and 65% at 1, 3, and 7 DAT, respectively. Bermudagrass, Kentucky bluegrass, and perennial ryegrass had 4, 4, and 2 metabolites after 7 d, respectively. Results suggest differential metabolism of flucarbazone–sodium is attributed to selectivity for controlling perennial ryegrass in bermudagrass and Kentucky bluegrass.
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