Abstract
Two recent studies (Cambier etal., 2017; Madigan etal., 2017) reveal invivo functions for specific phenolic glycolipids (PGLs) in the mycobacteria that cause tuberculosis or leprosy. M.tuberculosis (and M.marinum) PGL promotes bacterial spread to growth-permissive macrophages, while M.leprae PGL-1 induces macrophages to cause nerve demyelination characteristic of human leprosy.
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