Abstract
External root resorption (ERR) after orthodontic treatment represents one of its associated negative consequences. For several years, the existence of risk factors that contribute to the establishment of this alteration has been described, where we find genetic alterations related to the orthodontic treatment performed.
Highlights
The external root resorption (ERR) represents one of the iatrogenic effects related to the treatment of orthodontics either by the use of fixed apparatus or transparent aligners, where it predisposes to the loss of the root structure as is the cement or dentin [1,2,3,4]
Initial root resorption is caused by the activity of macrophage-like cells from the periodontal ligament, which causes damage to the root cement matrix, producing a denuded dentin root surface which is more susceptible to resorption as a result of osteoclast activity [5,6]
Diagnosis External root resorption is usually diagnosed as a clinical finding, when a routine x-ray is analyzed
Summary
The external root resorption (ERR) represents one of the iatrogenic effects related to the treatment of orthodontics either by the use of fixed apparatus or transparent aligners, where it predisposes to the loss of the root structure as is the cement or dentin [1,2,3,4]. Due to dental movement and as a consequence of sterile inflammation of the periodontal ligament and alterations in the microcirculation that allows the presence of a large number of cytokines and molecular factors involved with the activation of osteoclasts, including the kappa-B (Rankl) /Rank nuclear factor ligand receptor activator, osteoprotegerin (OPG) pathways and interleukin (IL-1). Initial root resorption is caused by the activity of macrophage-like cells from the periodontal ligament, which causes damage to the root cement matrix, producing a denuded dentin root surface which is more susceptible to resorption as a result of osteoclast activity [5,6]
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