Abstract
Crossed cerebellar diaschisis (CCD) is defined as functional mpairment due to decreased metabolism or blood flow in the cereellar hemisphere that is remote from, and contralateral to, the ausative supratentorial lesion. This widely known phenomenon is hought to be due to a transneuronal depression that is mediated y the corticopontocerebellar pathway. Crossed cerebellocerebral iaschisis (CCCD) is defined as the reverse of CCD, and has been uch less frequently described [1,2]. In fact, there are no reports n neuronal viability in CCCD. Here we report two cases in which e examined CCCD using 123I iomazenil single-photon emission omputed tomography (SPECT) in order to determine the neuronal iability.
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